- 1Laboratory of Neurogenetics, Molecular Neuropathology Section, National Institute on Aging, National Institutes of Health, Bethesda, MD, United States
- 2Department of Neurosciences, University of California, San Diego, La Jolla, CA, United States
- 3Division of Neuroscience, National Institute on Aging, National Institutes of Health, Bethesda, MD, United States
Citation: Iba M, Kim C, Florio J, Mante M, Adame A, Rockenstein E, Kwon S, Rissman R and Masliah E (2020) Role of Alterations in Protein Kinase p38γ in the Pathogenesis of the Synaptic Pathology in Dementia With Lewy Bodies and α-Synuclein Transgenic Models. Front. Neurosci. 14:286. doi: 10.3389/fnins.2020.00286
Received: 20 December 2019; Accepted: 12 March 2020;
Published: 31 March 2020; Retracted: 17 October 2025.
Edited by:
Cintia Roodveldt, Andalusian Center of Molecular Biology and Regenerative Medicine (CABIMER), SpainReviewed by:
Philipp Janker Kahle, Hertie Institute for Clinical Brain Research (HIH), GermanyYasuo Miki, Hirosaki University, Japan
Jazmin Florio2