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ORIGINAL RESEARCH article

Front. Neurosci., 31 March 2020

Sec. Neurodegeneration

Volume 14 - 2020 | https://doi.org/10.3389/fnins.2020.00286

RETRACTED: Role of Alterations in Protein Kinase p38γ in the Pathogenesis of the Synaptic Pathology in Dementia With Lewy Bodies and α-Synuclein Transgenic Models

Retracted

Michiyo Iba&#x;Michiyo Iba1†Changyoun Kim&#x;Changyoun Kim1†Jazmin FlorioJazmin Florio2Michael ManteMichael Mante2Anthony AdameAnthony Adame2Edward RockensteinEdward Rockenstein2Somin KwonSomin Kwon1Robert RissmanRobert Rissman2Eliezer Masliah,*Eliezer Masliah1,3*
  • 1Laboratory of Neurogenetics, Molecular Neuropathology Section, National Institute on Aging, National Institutes of Health, Bethesda, MD, United States
  • 2Department of Neurosciences, University of California, San Diego, La Jolla, CA, United States
  • 3Division of Neuroscience, National Institute on Aging, National Institutes of Health, Bethesda, MD, United States

Citation: Iba M, Kim C, Florio J, Mante M, Adame A, Rockenstein E, Kwon S, Rissman R and Masliah E (2020) Role of Alterations in Protein Kinase p38γ in the Pathogenesis of the Synaptic Pathology in Dementia With Lewy Bodies and α-Synuclein Transgenic Models. Front. Neurosci. 14:286. doi: 10.3389/fnins.2020.00286

Received: 20 December 2019; Accepted: 12 March 2020;
Published: 31 March 2020; Retracted: 17 October 2025.

Edited by:

Cintia Roodveldt, Andalusian Center of Molecular Biology and Regenerative Medicine (CABIMER), Spain

Reviewed by:

Philipp Janker Kahle, Hertie Institute for Clinical Brain Research (HIH), Germany
Yasuo Miki, Hirosaki University, Japan

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