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CORRECTION article
Front. Microbiol., 31 May 2018
Sec. Microbial Immunology
Volume 9 - 2018 | https://doi.org/10.3389/fmicb.2018.01168
This article is part of the Research TopicImmune Evasion Mechanisms in the Pathogenesis of Trypanosomatid InfectionsView all 50 articles
This article is a correction to:
Lipid Bodies as Sites of Prostaglandin E2 Synthesis During Chagas Disease: Impact in the Parasite Escape Mechanism
A corrigendum on
Lipid Bodies as Sites of Prostaglandin E2 Synthesis During Chagas Disease: Impact in the Parasite Escape Mechanism
by Almeida, P. E., Toledo, D. A. M., Rodrigues, G. S. C., and D'Avila, H. (2018). Front. Microbiol. 9:499. doi: 10.3389/fmicb.2018.00499
In our mini review article there was an error in the infected macrophage included in Figure 1A and the name of one of the authors of the Figure 1C was omitted.
Figure 1. Lipid bodies (LBs) biogenesis and components in both the host cell cytoplasm during the interaction and/or infection with T. cruzi and in the trypomastigotes forms of T. cruzi. (A) LBs accumulation (green) in murine infected macrophage after staining with BODIPY® 493/503. Nuclei of macrophage and internalized parasites were stained with DAPI (4′,6- diamidino-2-phenylindole; blue). (B) Schematic representation of the structural composition of a LB. Colored objects represent LBs surface-bound proteins located in the phospholipid monolayer. Prostaglandin E2 (PGE) 2, Arachidonic acid (AA), Diacilclycerols (DAG), Triacylglycerols (TAG) and cholesterol esters (CE) are found in the neutral lipid core. (C) Electron micrograph showing a LB in the trypomastigote form of T. cruzi. From: Melo, RCN (courtesy); Toledo, DAM and D'Avila, H.
The correct version of Figure 1A, its legend, and the name of the authors of Figure 1C appear below. The authors apologize for the mistake. This error does not change the scientific conclusions of the mini review article in any way.
The original article has been updated.
The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
Keywords: T. cruzi, lipid droplets, prostaglandin, infectious diseases, inflammation, lipid mediators, parasite replication, Chagas disease
Citation: Almeida PE, Toledo DAM, Rodrigues GSC and D'Avila H (2018) Corrigendum: Lipid Bodies as Sites of Prostaglandin E2 Synthesis During Chagas Disease: Impact in the Parasite Escape Mechanism. Front. Microbiol. 9:1168. doi: 10.3389/fmicb.2018.01168
Received: 07 May 2018; Accepted: 14 May 2018;
Published: 31 May 2018.
Edited and reviewed by: Celio Geraldo Freire-de-Lima, Universidade Federal do Rio de Janeiro, Brazil
Copyright © 2018 Almeida, Toledo, Rodrigues and D'Avila. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Heloisa D'Avila, ZGF2aWxhLmJpemFycm9AdWZqZi5lZHUuYnI=
Patrícia E. de Almeida, cGF0cmljaWEuYWxtZWlkYUB1ZmpmLmVkdS5icg==
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